Steroid research
This is a way for the body to selectively activate only certain genes. steroid research Steroid names. In this case, only those genes associated with androgens are activated, or have their activity increased. mRNA is different for each gene, and carries the information the cell needs to make specific proteins. Myosin and actin, which are major components of muscle, are examples of proteins, and these are made, ultimately, as a result of mRNA production from the genes for those proteins. steroid research Steroid fakes. At last: muscle protein, our goal. The molecule of AAS ultimately causes the muscle cell to make more of certain proteins, helping the user to get bigger. (There were steps needed to get from the mRNA to the protein, but we will skip them. steroid research Buying anabolic steroids. )Does each binding of AAS to an AR result in exactly one extra molecule of protein produced? No. Because even though the AR is fully activated by any AAS, that does not mean that it always succeeds in binding to DNA. And differing amounts of mRNA might be produced, because an AR remains active as long as an AAS remains bound to it. If many mRNA molecules are produced, then, generally, they will cause many corresponding protein molecules to be produced. So the amount of extra growth per extra activated AR can vary. The Androgen ReceptorNow, having a broad view of the process, let's take a closer look at the AR itself. The AR is a large protein molecule, produced from one and only one gene in DNA. There aren't lots of different kinds of receptors, as some authors claim. There are not, for example, ARs specific for oral or injectable anabolics, nor for different esters of testosterone, nor for any different kinds of AAS. The first important question to ask is, "How many ARs do you have? Is the number small or large? Can it be changed?" Since these are, in effect, little machines which are either on or off, and their effect is greater as more are activated, we want as many of them switched on as possible. There are far fewer ARs than most people realize. Some authors who are opposed to AAS doses beyond 200 mg/week say that only this amount will be accepted by the receptors in muscle, and everything past that will "spill over" and go into receptors in the skin and elsewhere. Research shows that muscle tissue has, roughly, 3 nanomoles of ARs per kg. Then your body probably has less than 300 nanomoles of ARs, grand total, let's say. Well, one 2. 5 mg tab of oxandrolone supplies about 8000 nanomoles of AAS. Clearly, that's far more molecules than your body has receptors. A little math shows that all those receptors combined could bind only a small percentage of the molecules of AAS in one little 2. 5 mg tab. So binding to ARs cannot appreciably reduce the concentration of AAS in the blood. Therefore, the ideas that ARs will bind most of whatever dose some author recommends, or that "spill-over" will occur beyond that, are entirely wrong.
Steroid research
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