Anabolic discussion boards
Generally, the errors are not unique to WAR, but are commonly believed, and furthermore, the correct information is of value. anabolic discussion boards Steroids. Anabolicum Vister (Quinbolone)This oral anabolic/androgenic steroid (AAS) is actually a prodrug of boldenone. It is the exact same molecule, but with a cyclopentenyl ether modification at position 17 (recall that injectable AAS often use ester modifications at 17). This allows for some degree of oral effectiveness. anabolic discussion boards I need to know the effects of the steroid winstrol. The prodrug is not active until it is converted to the parent drug in the body, by removal of this group. Quinbolone is not very potent (effective per milligram) compared to injected boldenone undecylenate (Equipoise), which itself is not particularly potent. Contrary, however, to the claim in WAR, this compound should aromatize comparably to Equipoise, based on its chemical structure. anabolic discussion boards Steroids. However, I know of no athletes who use it, so I cannot say that this is demonstrated. Anadrol (Oxymetholone)Although this AAS can indeed lead to estrogenic side effects, it is very questionable, as Pat Arnold has pointed out, if it itself converts to estrogen at all. There is no evidence in the scientific literature for that, and if it does do so, it is not by a mechanism known for other steroids. Aromatase would be required but would not be sufficient. It may be the case that oxymetholone affects estrogen metabolism, perhaps either by slowing its rate of elimination, or by upregulating aromatase production. Whatever the cause, antiestrogens should be employed with this drug unless the user simply does not care about estrogenic side effects. Oxymetholone does not convert to DHT. However, it is a potent androgen in the skin and scalp nonetheless. Use of a 5-alpha reductase inhibitor, such as Proscar, would be of no use, since the compound cannot be 5-alpha reduced. Anadur (Nandrolone hexylphenylpropionate)There is no support for the claim that nandrolones require more protein intake than is needed with other AAS. There is, in my opinion, a tendency on the part of the WAR authors to see a statement concerning a particular steroid, and if they have not seen it for other steroids, to then assume that the property is unique to that steroid. Here, because the information packet with the drug says that the patient must be sure to consume sufficient protein, they imagine this is peculiar to the drug. It is, however, true for all AAS. The claim that aromatization is less than with Deca Durabolin is unlikely to be correct. Because of the longer half life, for the same dose, there will be less drug in the blood during the first few weeks of use, thus creating this illusion. If blood nandrolone levels are the same, giving comparable effectiveness, rate of aromatization would be the same. This is true for all claims made for varying rates of aromatization according to ester. I do not think such claims are ever correct given equal blood levels of drug, and they certainly are not substantiated in the scientific literature. The claim that 50-100 mg per 10 days will normally result in no virilization symptoms for women will be over-optimistic for many female users. Only a few weeks of such use will result in some irreversible virilization for some women.
Anabolic discussion boards
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