Sternum pain
There are two popular theories regarding the pathogenesis of rheumatoid arthritis (RA). sternum pain Toothache pain relief. The first holds that the T cell, through interaction with an - as yet unidentified - antigen, is the primary cell responsible for initiating the disease as well as for driving the chronic inflammatory process. This theory is based upon the known association of RA with class II major histocompatability antigens, the large number of CD4+ T cells and skewed T cell receptor gene usage in the RA synovium. The second theory holds that, while T cells may be important in initiating the disease, chronic inflammation is self-perpetuated by macrophages and fibroblasts in a T-cell independent manner. sternum pain Arthritis cartoons. This theory is based upon the relative absence of activated T cells phenotypes in chronic RA and the preponderance of activated macrophage and fibroblast phenotypes. Histopathology Synovium Cartilage Bone Synovial Cavity Synovium The synovium, in normal joints, is a thin delicate lining that serves several important functions. (Slide) The synovium serves as an important source of nutrients for cartilage since cartilage itself is avascular. sternum pain Enbrel arthritis. In addition, synovial cells synthesize joint lubricants such as hyaluronic acid, as well as collagens and fibronectin that constitute the structural framework of the synovial interstitium. 1. Synovial lining or intimal layer: Normally, this layer is only 1-3 cells thick (slide). In RA, this lining is greatly hypertrophied (8-10 cells thick) (slide).
Sternum pain
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