Anabolic steroid cutting cycles
Now if all AAS behaved the same way and differed only in their potencies, and had the same ratios of potency regardless of the activity being studied (whether in muscle or skin or nerves, etc. anabolic steroid cutting cycles Calculate ideal weight. ) then this would be consistent with there being only one target or receptor. However, if some AAS are effective in some activities but do nothing in others, while other AAS do have these other activities, then this can't all be occurring from the same receptor. Most of the research in this area is rather far removed from bodybuilding, but the principles still apply. anabolic steroid cutting cycles Free bodybuilding diet. Biochemistry is usually much broader than any one specific cell being studied. (For example, most human biochemistry was actually learned originally by study of E. coli and with later research found to be identical in man. anabolic steroid cutting cycles Human-anatomy-muscles. ) Thus, while we may not care about ductal branching morphogenesis in the developing rat prostate, the fact that a peculiar biochemical mechanism of androgen response occurs here implies that such a mechanism may well exist in things we are interested in, such as bodybuilding. The possibility at least exists. Speaking of ductal branching morphogenisis in the developing rat prostate,6 here indeed different steroids behave differently. While to the AR testosterone is less potent than DHT, here the reverse relationship was found. Furthermore, methyltrienolone, which is a more potent agonist (activator) of the AR than is DHT, was no more effective than DHT in inducing ductal branching and was less effective than testosterone. This cannot be explained by assuming that aromatization of testosterone to estradiol contributed to the process, because 5a -androstan-3a ,17b -diol (which cannot aromatize) was similarly potent. Thus, there is some target or receptor in these tissues which has different "preferences" (Kd values, and different rank order of potency) than the AR does. Could this also be the case for muscle growth? Perhaps. Another example is found in the virilization of the mammary gland of female rats. 7 The same results are seen here as in the above example of the rat prostate. Testosterone (T) has more activity than DHT does, though at the AR that would not be so. Differences also are seen in the male accessory glands of the rabbit and rat. 8 Testosterone propionate and DHT propionate were found to be equally potent in supporting growth and secretory activity of these glands, but the above-mentioned 5a -androstan-3a ,17b -diol was considerably more potent than these in the prostate but completely ineffective in the epidydimis. Furthermore, use of an antiandrogen (AR blocker) did not affect the function of the epidydimis at all. Thus, the activity of testosterone and DHT in this tissue is not via the AR.
Anabolic steroid cutting cycles
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