Steroid comparison chart
Differences Between the Antiestrogens, Anti-aromatases versus Estrogen Antagonists and the Practical Use of Cytadren and Clomidby Bill RobertsBill is the formulator of Biotest's MAG-10, the first and only legal androgen delivery system that produces outstanding muscular gains through the combination of A1E and 4-AD-EC - the premier legal Class I and Class-II androgens. steroid comparison chart Anabolic steroids sports. Read MAG-10: A New Orally Effective Androgen for additional information regarding the synergestic effects of Class I and Class II androgens. Chemical Muscle EnhancementChemical Wizardry by George Spellwin - Definitive Anabolic Steroid Database The Layman's Guide to Steroids - Mick Hart's Best-Selling Anabolic Steroid GuidesLegal Muscle by Rick Collins: Anabolics in AmericaMuscle Building Nutrition by Will Brink - Serious Lean Muscle Gains without the BodyfatSteroids 101 by Jeff Summers - The Book That Makes It Fun To Learn About Anabolic SteroidsBurn the Fat, Feed the Muscle by Tom Venuto - Fat Burning Secrets of the World's Best Bodybuilders and Fitness ModelsAnabolics 2002Publication Date: June 1998Nothing in this article is intended to take the place of advice from a licensed health professional. Consult a physician before taking any medication. steroid comparison chart Steroid pictures. What does "anti-estrogen" mean? How are anti-estrogens like Cytadren, Clomid, and Nolvadex different from each other? Is Proviron an anabolic steroid, or not?Anti-estrogens are drugs which act to reduce estrogenic activity in the body. This can be done either by reducing the amount of estrogen, or by reducing the activity of whatever estrogen is present. Competitive aromatase inhibitors, such as Cytadren, Arimidex, and probably Proviron, bind to the same binding site on the aromatase enzyme that testosterone does. steroid comparison chart Anabolic steroid cycle. By doing this, they allow less testosterone to bind to aromatase. So, less testosterone is converted to estradiol (estrogen). Here's an important thing: the effectiveness of competitive inhibitors decreases as the amount of the normal substrate increases. Suppose that you had equal amounts of inhibitor and normal substrate in the blood, and they bound to the enzyme equally well. Then the inhibitor would at any moment be taking up half the sites that the normal substrate otherwise would, so it would reduce conversion rate by 50%. But if the amount of substrate is increased 10 times while the amount of inhibitor remains the same, then the inhibitor would be outcompeted by the more numerous substrate molecules. It would therefore be rather ineffective. For example, with more testosterone molecules available, and similar binding strengths, the enzyme will mostly bind testosterone. It will then mostly be working to produce estrogen. To obtain the 50% reduction we had before, then the amount of inhibitor would also have to be increased 10 times. To be really effective, the inhibitor must either be present in higher concentration than the normal substrate, or must bind more tightly. With Cytadren or Proviron, it takes quite a lot of inhibitor to outcompete high testosterone levels. With Arimidex, rather little, even 1 mg/day, can be sufficient because it binds so strongly. The other general approach is estrogen receptor antagonism. If a molecule binds strongly to a hormone receptor, but does not activate that receptor and makes it unresponsive to the normal hormone, then it is a receptor antagonist. Clomid (clomiphene) and Nolvadex (tamoxifen) follow this approach.
Steroid comparison chart
Anabolics || Controversial on steroids || Buying steroids online || Buying steroids