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I suspect that it not only acts as an antiaromatase but in an unknown DHT-like anti-estrogenic manner. steroid board Athletes and steroids. This might involve estrogen receptor downregulation for example. In any case, aromatase inhibition and/or Clomid don't seem to give the same effect on appearance and muscle hardness as when Proviron is included. How much of these agents is needed for effective estrogen suppression?Again, it depends on the dose of anabolic/androgenic steroids (AAS) and it depends what type of AAS is being used. steroid board Athletes and steroids. With Primobolan or trenbolone there is no need for these drugs. With nandrolone, an aromatase inhibitor will be of no use, because aromatase is not used in the aromatization of nandrolone. A rather small amount of estrogen receptor antagonist can be useful. steroid board Steroid board. 12. 5 to 25 mg Clomid would be plenty for 400 mg/week Deca. With testosterone, stacking of an aromatase inhibitor and an estrogen receptor antagonist will give the best results. Cytadren use should not exceed 250 mg/day in my opinion. This alone would not be sufficient for say 1 g/week or more of testosterone. With such a dose, ideally one would add in 50 mg/day Clomid. Proviron at 100 mg/day could substitute for the Cytadren. Or Cytadren and Proviron can be used in combination, 125/50 or higher, together with 50 mg/day Clomid. For lower doses of testosterone, proportionally less antiestrogens can be used. Arimidex is very effective but extremely expensive. 1 mg/day of this is at least as effective as 250 mg/day Cytadren. If a milligram per day cannot be afforded, use of half a milligram would allow Cytadren use to be cut in half, which may be desirable. How does Clomid "stimulate" testosterone production at the end of the cycle?It really doesn't. Rather, by acting as an estrogen receptor antagonist, it reduces the inhibition that results from elevated estradiol levels. This helps return LH to normal levels, which helps testosterone to return to normal levels (if the testicles have not atrophied). How does hCG help?Acts as an LH receptor agonist, thus substituing for LH. It does nothing to help the hypothalamus and pituitary. Thus, it can be effective during the cycle to help avoid testicular atrophy, but is not best used in the taper when one is attempting to restore LH production. Increases in natural testosterone, stimulated by the hCG, will act to inhibit LH production. Thus, you can see where hCG use is counterproductive in the taper itself. Can Clomid, taken throughout a cycle, completely eliminate inhibition?I do not believe so. There is also androgenic inhibition mediated by the androgen receptor, which has nothing to do with the estrogen receptor.

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