Sports steroids
The dosing schedule given (two days on, two days off) makes no sense because the half life of the drug is long enough that the "off" days still have high levels of drug in the system: thus receptor desensitization will not be avoided. sports steroids Where do steroids come from. Aside from that theoretical reason, it is also observation that the scheme simply does not work to prolong the effectiveness of the drug. Clomid (Clomiphene citrate)Clomid's activity is achieved not by stimulation of the hypothalamus, but by blocking inhibition of the hypothalamus by estrogen. Clomid is a mixed estrogen agonist/antagonist (activator/blocker) not because it is a "weak estrogen" but because it puts the estrogen receptor in a somewhat different conformation (shape) than does estradiol. sports steroids Sports steroids. The result is that in some tissues Clomid acts as an antagonist, and in others as an agonist, depending on what cofactors are used in that tissue in combination with the estrogen receptor. Clomid is an effective antagonist in the hypothalamus and in breast tissue. It is an effective agonist in bone tissue, and for improving blood cholesterol. sports steroids Steroid profiles dosages. The claim that duration of intake should not exceed 10-14 days is incorrect. Clinical studies with male patients have been for periods of a year or longer. This error probably originates from the fact that, for use in women, due to the menstrual cycle there would obviously be no point in trying to stimulate ovulation all four weeks of the month. Thus, use in women is limited to 10-14 days. That limitation is not because of toxicity. Clomid is in fact useful throughout a cycle if aromatizable drugs are being used. Cytadren (Aminoglutethimide)Cytadren, at moderate doses, is a fairly effective inhibitor of aromatase and a weak inhibitor of desmolase (an enzyme needed for the production of all steroids), and at higher doses becomes an effective inhibitor of desmolase. Inhibition of desmolase will lead, at least temporarily, to decreased production of cortisol. Contrary to the claim in WAR, there is no evidence, nor good reason to believe, that reducing cortisol below normal would be of benefit to the weight training athlete, and considerable evidence that it is a bad idea. Thus, desmolase inhibition is to be avoided. The claim that Cytadren significantly inhibits natural production of testosterone is not correct. The desmolase step is rate limiting for the production of cortisol, but not for testosterone: thus, a slowing of desmolase activity does not slow production of testosterone significantly. However, the conclusion that natural athletes should not use Cytadren is correct, but for a different reason. Natural athletes have no need of the antiaromatase activity, and the anti-desmolase activity, reducing cortisol below normal, is not desirable. It would only be desirable if cortisol levels were abnormally high, which should not be the case. The claim that 2-4 tablets per day (500-1000 mg) should be taken is extraordinarily bad advice. I do not believe that more than 250 mg/day should be taken, and that should be taken very carefully, divided into 125 mg (half a tab) in the morning, and 62. 5 mg (quarter tab) six and twelve hours after that. The two on, two off idea is also not a good one, since that gives no antiaromatase activity half of the time. It has been demonstrated that 250 mg/day is not much less effective than 1000 mg/day in inhibiting aromatase, but the smaller dose results in much less desmolase inhibition. If the drug is suddenly discontinued, cortisol rebound may occur. Thus, the drug should be tapered down. Deca-Durabolin (Nandrolone Decanoate)Deca has no peculiar property in terms of enhancing protein synthesis besides that which all AAS have. The claim that it does not aromatize at doses below a given threshold (400 mg/ week is stated) is not correct. It aromatizes at any dose, but at a lower rate than testosterone does.
Sports steroids
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