CEREBRAL PALSY CAUSES
WHAT CAUSES CEREBRAL PALSY?
We do not know the cause of most cases of cerebral palsy, but many have been to 'Black or Red palaces" means celebrations or funeral according to all the data's of the parents between 75%.There are a lot of data that show that during pregnancy the mother has had some form of bad or horrified experience or have had been to some kind of ceremony of the un present once 60%.
Still we are unable to identify, we are unable to determine what caused cerebral palsy in most children who have congenital CP. We do know that the child who is at highest risk for developing CP Cerebral Palsy is the premature, very small baby who does not cry in the first five minutes after delivery, who needs to be on a ventilator for over four weeks, and who has bleeding in his brain (heamorrage). Babies who have congenital malformations in systems such as the heart, kidneys, or spine are also more likely to develop CP- Cerebral Palsy, probably because they also have malformations in the brain.
Cerebral Palsy Parents- When a diagnosis of CP is made, the mother and father often feel guilty and wonder what they did to cause their child to have this disorder. While it is certainly true that good prenatal care is an essential part of preventing congenital problems, it must be stated that congenital problems, or "birth defects," often occur even when the mother has strictly followed her physician's advice in caring for herself and the developing infant.
When the fetus is exposed to certain chemicals or infections through the expectant mother, for example. The developing brain can be injured if the expectant mother suffers severe physical trauma, the fetal brain can be injured, too, but this is rare CEREBRAL PALSY.
Finally, pre maturity and a low birth weight have been shown to be related to an increased incidence of specific disorders like cerebral palsy. Many chemicals are known to adversely affect the developing brain, alcohol being the most commonly used. The term Fetal Alcohol Syndrome describes the long-term, multi-system effect of alcohol on a child whose mother abused alcohol during the pregnancy. When a fetus is exposed to large amounts of alcohol, several body systems, including the neurological system will almost certainly suffer damage.
Cigarette smoking by the mother has been shown to decrease birth weight, and low birth weight is associated with several disorders, including cerebral palsy. Severe malnutrition in the mother can adversely affect brain growth in the fetus, and it, too, can result in a low birth weight.
The use of cocaine or crack by the expectant mother is associated with blood vessel complications, and these complications affect many organs as well as the central nervous system. Cocaine use is increasing and thus becoming more prevalent as cause of brain damage in infants. Most infants whose mothers used cocaine during pregnancy develop mental retardation rather than cerebral palsy, however. Infections such as rubella (German measles), toxoplasmosis, and cytomegalovirus (CMV), ( if a woman has them during pregnancy), also may injure the brain of the fetus. Rubella can be prevented by immunization, prior to becoming pregnant, and the chances of becoming infected with toxoplasmosis can be minimized by not handling the feces of cats and by avoiding raw or uncooked meat.
Congenital infection with human immunodeficiency virus (HIV, the virus that causes AIDS) also causes brain damage in children, though it usually causes mental retardation rather than CP.
It is likely that many other infections in the expectant mother injure the developing fetus, but they are not recognized as causative factors because the woman who has the infection either does not recognize the symptoms of infection or is symptom-free. Premature infants are at a much higher risk for developing cerebral palsy than full-term babies, and the risk increases as the birth weight decreases. Between 5 and 8 percent of infants weighing less than 1500 grams (3 pounds) at birth develop cerebral palsy, and infants weighing less than 1500 grams are 25 times more likely to develop cerebral palsy than infants who are born at full term weighing more than 2500 grams.
Any premature infants suffer bleeding within the brain, called intraventricular hemorrhages, intracranial hemorrhages. Again, the highest frequency of hemorrhages is found in the babies with the lowest weight: the problem is rare in babies who weigh more than 2000 grams (4 pounds).
This bleeding may damage the part of the brain that controls motor function and thereby lead to cerebral palsy. If the hemorrhage results in destruction of normal brain tissue (a condition called periventricular leukomalacia) and small cysts around the ventricles and in the motor region of the brain, then that infant is more likely to have CP than an infant with hemorrhages alone.
It also explains why modern obstetrical care, including monitoring and a high rate of Cesarian section, has lowered infant mortality rates but not the incidence of cerebral palsy. One large study, for example, has shown that more than 60 percent of all pregnancies have at least one complication, and that most of these complications cause no problems. For instance, 25 percent of all newborns have the umbilical cord wrapped around their neck, and 16 percent passed meconium (had the first bowel movement) at the time of birth.
These "birth events" and the development of CP have only a small correlation. In other words, the chances of a child developing CP were nearly the same whether the child was born with a cord wrapped around her neck or not. On the other hand, newborns in this study who had very low Apgar scores (less than 3 at 20 minutes) had a risk 250 times greater than infants with normal Apgar scores of developing cerebral palsy.
An Apgar score at this level suggests that the infant suffered severe asphyxia (lack of sufficient oxygen to the brain) during birth. Half of the infants who suffered severe asphyxia during birth did not develop cerebral palsy, however. When CP is diagnosed in childhood, it is often discovered that the child suffered asphyxia at birth, but the asphyxia is usually considered the symptom of an otherwise sick baby with a neurological problem, and not the primary cause of CP.
In two different large studies, only about 9 percent of children with CP- Cerebral Palsy were thought to have CP directly and exclusively related to asphyxia at delivery. Ninety-one percent of the babies had other inherent causes which led to pre maturity or perinatal or neonatal problems (problems In the nineteen century, Dr. William John Little described cerebral palsy and stated that the condition was due to birth injury in most cases.
One such injury is asphyxia, which can damage the brain in a variety of ways, and is the number one cause of CP Cerebral Palsy in this age group. The three most common causes of asphyxia in the young child are: choking on foreign objects such as toys and pieces of food (including peanuts, popcorn, and hot dogs); poisoning; and near drowning. The brain may also be damaged when it is physically traumatized as a result of a blow to the head. A child who falls or is involved in a motor vehicle accident or is the victim of physical abuse may suffer irreparable injury to the brain.
One form of child abuse is the shaken baby syndrome, in which the caretaker is trying to quiet the baby by shaking too vigorously, causing the brain to strike repeatedly against the skull under high pressure.
Severe infections, especially meningitis or encephalitis, can also lead to brain damage in this age group. Meningitis is inflammation of the meninges ( the covering of the brain and the spinal cord), usually caused by a bacterial infection, and encephalitis is brain inflammation which may be caused by bacterial or viral infections.
Either of these infections can cause disabilities ranging from hearing loss to CP Cerebral Palsy to severe retardation which all can be treated with Acupuncture and Herbal Treatment Centre in KL Chinese Master's CEREBRAL PALSY NEURO ACUPUNCTURE AND BRAIN POWDER HERBAL MEDICINE.
Children with spinal cord dysfunction, for example, face medical problems such as insensate skin and bowel and bladder dysfunction, which differ markedly from the medical problems faced by children with cerebral palsy. Spinal cord dysfunction may be a result of spinal cord injury, spina bifida (meningomyelocele), or a congenital spinal cord malformation.
Another large group of children who at time may look similar to those with cerebral palsy are children with temporary motor problems resulting from closed head injuries, seizures, drug overdoses, or some brain tumors.
The medical issues for this group of children are also different from the medical issues for children with cerebral palsy, because these injuries can occur at any age and the severity of the problems caused by these injuries changes over time. We can also say that disorders that are primarily of muscle, nerve, and bone are not cerebral palsy by definition. Such conditions include muscular dystrophy, peripheral neuropathies such as Charcot-Marie- Tooth disease, and osteogenesis imperfecta.
All of these "Brain Damage Cerebral Palsy "conditions are associated with specific medical problems. Children with progressive neurologic disorders (including Rett's syndrome, leukodystrophy, and Tay-Sach's disease) also have medical needs which are different from those of children with cerebral palsy.
Some children with chromosomal anomalies (for example, trisomy 13 and 18) or congenital disorders (hereditary spastic paraplegia, for example) may appear similar to children with cerebral palsy; others, such as children with Down's syndrome, appear very different from children with cerebral palsy. Children with these disorders have some problems in common with children who have cerebral palsy; they also have problems that are unique for children with that specific disorder.
A physician may suspect cerebral palsy in a child whose development of these skills is delayed. In making a diagnosis of cerebral palsy, the physician takes into account the delay in developmental milestones as well as physical findings that might include abnormal muscle tone, abnormal movements, abnormal reflexes and persistent infantile reflexes.
Making a definite diagnosis of cerebral palsy is not always easy, especially before the child's first birthday. In fact, diagnosing cerebral palsy usually involves a period of waiting for the definite and permanent appearance of specific motor problems. Most children with cerebral palsy can be diagnosed by the age of 18 months, but eighteen months is a long time for parents to wait for a diagnosis, and this is understandably a difficult period for them. Making a diagnosis of cerebral palsy is also difficult when, for example, a two-year- old has suffered a head injury. The child may immediately appear to be severely injured, and three months after the injury he may have symptoms that are typical of a child with cerebral palsy. But one year after the injury such a child may be completely normal. This child does not have cerebral palsy. Although he has a scar on his brain, the scar is not permanently impairing his motor activities. After injury, waiting and observing are necessary before the diagnosis can be made. Diagnosis of cerebral palsy,examination is the physical evidence of abnormal motor function. A diagnosis of cerebral palsy cannot be made on the basis of blood test, though the physician may order such tests to exclude other neurologic diseases (such as those mentioned above).
Cerebral Palsy Type
To avoid confusion, most of the discussion will be limited to the use of these three terms. Occasionally such terms as paraplegia, double hemiplegia, triplegia, and pentaplegia may occasionally be encountered ; these classifications are also based on the parts of the body involved in each Cerebral Palsy Case. The dominant type of movement or muscle coordination problem is the other method by which children are subdivided and classified to assist in communicating about the problems of cerebral palsy. The component which seems to be causing the most problem is often used as the categorizing term. For example, the child with spastic diplegia has mostly spastic muscle problems, and most of the involvement is in the legs, but the child may also have a smaller component of athetosis and balance problems. The child with athetoid quadriplegia, on the other hand, would have involvement of both arms and legs, primarily with athetoid muscle problems, but such a child often has some ataxia and spasticity as well. Generally a child with quadriplegia is a child who is not walking independently.
The reader may be familiar with other terms used to define specific problems of movement or muscle function terms such as: dystonia, tremor, ballismus, and rigidity.
The words severe, moderate, and mild are also often used in combination with both anatomic and motor function classification terms (severe spastic diplegia, for example), but these qualifying words do not have any specific meaning.
Usually asked by parents after they are told their child has cerebral palsy are "What will my child be like?" and "Will he walk?" Predicting what a young child with cerebral palsy will be like or what he will or will not do (called the prognosis) is very difficult. Any predictions for an infant under six months of age are little better than guesses, and even for children younger than one year it is often very difficult to predict the pattern of involvement. By the time the child is two years old, however, the physician can determine whether the child has hemiplegia, diplegia, or quadriplegia OF CEREBRAL PALSY.
Based on this involvement pattern, some predictions can be made. It is worth saying again that children with cerebral palsy do not stop doing activities once they have begun to do them. Such a loss of skills, called regression, is not characteristic of cerebral palsy. If regression occurs, it is necessary to look for a different cause of the child's problems. In order for a child to be able to walk, some major events in motor control have to occur. A child must be able to hold up his head before he can sit up on his own, and he must be able to sit independently before he can walk on his own. It is generally assumed that if a child is not sitting up by himself by age 4 or walking by age 8, he will never be an independent walker. But a child who starts to walk at age 3 will certainly continue to walk and will be walking when he is 13 years old unless he has a disorder other than CP. Difficulty to make early predictions of speaking ability or mental ability than it is to predict motor function. Here evaluation is much more reliable after age 2, although a motor disability can make the evaluation of intellectual function quite difficult.
Sometimes "motor-free" tests which can assess intellectual ability without, the person being tested, needing to use his hands are administered by psychologists who have expertise in their use. Overall, the intellectual ability of the person, far more than their physical disability, will determine the person's prognosis. In other words, mental retardation is far more likely than cerebral palsy to impair a child's ability to function.
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Parents are naturally concerned when their newborn child has problems, and physicians need to evaluate the child's condition and prognosis as well as they can. For example, evidence of a bleed in the child's brain should be discussed with parents, although the outcome of such a bleed cannot be predicted.
The diagnosis of cerebral palsy cannot be made at birth and, most assuredly, the extent and severity of involvement that an individual child might eventually have is impossible to assess at birth.
Many neonatologists, aware of the interaction that generally occurs between the newborn and parents, avoid discussing the child's problems in detail because they want to permit this interaction to take place. The presumption of a bleak future for a child sometimes causes parents to withdraw from the child and this can have a significant negative effect on the child. Physicians usually communicate their concerns in terms of the child's symptoms, such as muscle problems, and prepare parents for the possibility of neurologic damage. Clearly, it is part of the physician's role to inform parents, but the variability of outcome makes it virtually impossible for the physician to predict the future, and so the physician must weigh the need to inform (and the imprecision of information) against the need for the parents to have hope for, and to become close to their child.
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